Hyperphosphatemia can lead to muscle cramps and tetany due to its effect on calcium levels.

Close monitoring of electrolyte levels is crucial for detecting and managing the imbalances associated with TLS.

Insulin with dextrose is used to shift potassium from the bloodstream into the cells, temporarily reducing serum potassium levels.

IV fluids help flush out the released intracellular contents, such as uric acid and electrolytes, through the kidneys, reducing the risk of renal damage.

Trousseau's sign is a clinical indication of hypocalcemia, which is a common electrolyte disturbance in TLS.

Elevated serum creatinine indicates renal impairment, which is a common complication of TLS due to the precipitation of uric acid in the kidneys.

Allopurinol is commonly used to prevent and treat hyperuricemia in patients with TLS by inhibiting the formation of uric acid.

Hyperkalemia and other electrolyte imbalances in TLS can lead to dangerous cardiac arrhythmias.

TLS often leads to hyperkalemia due to the release of potassium from lysed tumor cells.

TLS occurs when a large number of tumor cells are rapidly destroyed by chemotherapy, releasing their intracellular contents into the bloodstream.